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Description

Pathogenic alpha-synuclein aggregates preferentially bind to mitochondria and affect cellular respiration

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The F(ab)' 2 may be reduced under mild conditions to break the disulfide linkage in the hinge region thereby converting the (Fab') 2 dimer into a Fab' monomer

This pathway is implicated in BDNF-mediated neuronal differentiation [96], survival [97], neurite outgrowth of dopaminergic neuron [98], NMDA receptor phosphorylation [99], AMPA receptor phosphorylation [100], -aminobutyric acid (GABA) receptor transcription [101], GABA-A receptor internalization during memory consolidation [102], spinal motor potentiation [103], and hippocampal activity integration and plasticity facilitation [104]