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and (iii) inhibition is exerted by binding to the NADP + -TGR(H) reduced species

Lipid uptake Tumor cells undergo significant lipid metabolic reprogramming to compensate for glucose shortages, resulting in excessive lipid accumulation within the tumor microenvironment (TME) [40]

IRP1 and IRP2 are cytosolic proteins that bind to IRE in the mRNA of different target genes and control their translation, including TfR1, DMT1, ferritin, and FPN1 [79]

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