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Pulukuri, S

coli has been documented, and it has been determined that this resistance is caused by a mutation in the gene that codes for the oxygen-insensitive enzyme nfsA

564 Studies indicate that hepatic PPAR expression contributes to steatosis by promoting triglyceride synthesis and impairing glucose tolerance through monoacylglycerol O-acyltransferase 1 (MGAT1)

This results in NRF2 accumulation and CDDP resistance [105]

However, the exact therapeutic mechanism remains elusive, and further prospective studies are needed to confirm the effectiveness of MSCs in GVHD treatment

15 , 16 Bioinformatic analysis offers a powerful tool for identifying disease correlations and molecular mechanisms