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[DOI] [PMC free article] [PubMed] [Google Scholar] Roumes, H

In other contexts where metabolic support is provided to retinal ganglion cells in glaucoma, inflammation persists ( e.g

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(52) demonstrated that potassium two pore domain channel subfamily K member 1(KCNK1) promotes histone Kla by binding to and activating LDHA, which in turn increases glycolysis and lactate generation in BC cells

The absence of apoptosis in Spint1a-deficient larvae, together with the ability of Parp inhibitors to robustly reduce keratinocyte cell death in this model, led us to hypothesize that Parp1 overactivation in response to extensive ROS-mediated DNA damage would promote parthanatos cell death