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As was mentioned before, ROS1 fusions in melanoma and favor melanoma lesions are rare and include: GOPC::ROS1 in acral lentiginous conventional melanoma [109] and a case of unresectable Spitz tumor [105] with dramatic response to ROS1 targeting tyrosine kinase inhibitorsentrectinib and crizotinib in both cases

Given the growing understanding of the regulatory networks that drive phenotype switching and its role in melanoma metastasis and therapy resistance, one strategy is to block or reverse the invasive switch by directing melanoma cells towards the more therapy-sensitive proliferative/melanocytic state [67]

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