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wherein: Q is a glutamine residue, wherein the glutamine residue is part of a Q-tag peptide sequence

[DOI] [PubMed] [Google Scholar] 149.Francisco JA, Cerveny CG, Meyer DL, et al

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The goal was to identify potential therapeutic targets and develop innovative treatment strategies for NAFLD/NASH, thereby transforming our approach to HCC prevention in the context of NAFLD/NASH

However, our expectations need to be experimentally supported in the future

The intensity of mTORC1 signaling appears to have consequences on differentiation