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doi: 10.1172/jci.insight.187072 97 LiuJLiKCaiJZhangMZhangXXiongXet al
Boston: William Andrew Publishing
A key driver behind this cellular hyperexcitability is a specific ion channel called HCN2
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Lipid metabolism Elevated ROS activate AMPK, which phosphorylates and inhibits acetyl-CoA carboxylase (ACC), reducing malonyl-CoA levels and thereby relieving the inhibition of carnitine palmitoyltransferase 1 (CPT1), the rate-limiting enzyme in fatty acid oxidation (FAO)
105134 (Springer, 2014)
