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Neurological deficits were detected 3 and 7 days after ICH

GSTP1 levels are associated with multiple human pathologies including multiple aggressive cancers, and over-expression is known to result in drug resistance [7]

However, the preceding sections of this paper establish that GSH is not merely a passive antioxidant, but an immune regulator and modulator with actions dependent upon the pathophysiologic context (Ghezzi, 2011) GSH acts to regulate the thresholds necessary for immunologic activation through redox-sensitive signal transduction pathways, influences antigen presentation and the development/differentiation of immune cells, and modulates the production of cytokines

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These oxidation reactions are triggered by environmental compounds that can cause cellular damage, potentially leading to health concerns