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To identify the ubiquitination machinery responsible for the turnover of FAD-free FSP1, we conducted a genetic screen in FSP1 GFP-P2A-BFP reporter cells, in which we deleted RFK to disrupt FAD synthesis

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Animal experiments utilizing various medications and formulas of Se, as well as trials in GPx mouse models, reported an important role in neutralizing reactive oxygen and nitrogen molecules, ultimately restricting organ injury following myocardial reperfusion

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Osteosarcopenia in rheumatoid arthritis treated with GCs occurs in 37.1% of patients with rheumatoid arthritis, and the incidence of sarcopenia increased with age: from 14% in patients aged 4049 years to 78,6% in patients aged 8089 years [54]