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The key step in the induction of mutagenic effects by Cr(VI) has been found to be the formation of Cr(III)-containing complexes between DNA and proteins [74,75]

Though the cause of ME/CFS is unknown, research indicates that 60-70% of cases are associated with viral infections (e.g., Epstein-Barr virus, H1N1, human herpesvirus, etc.).27 But the persistent symptoms experienced by long COVID patients are so similar to those seen in ME/CFS that some experts have proposed that long COVID may actually be another post-viral example of ME/CFS instead of a separate condition.28 While there appear to be a few different subtypes of ME/CFS,29 30 it stands to reason that patients with cases caused by COVID, or those most like long COVID, may see a similar restoration of an appropriate response from TA-1 peptide therapy as noted in the aforementioned ex vivo study.26 However, as with long COVID, far more research is needed to evaluate TA-1 as a potential treatment for ME/CFS

A Japanese clinical study (120) showed that in a phase I trial of sunitinib combined with s-1 and cisplatin in patients with GC, the MTD of sunitinib combined with cisplatin/S-1 was 25 mg/day