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In models of Alzheimer's disease, evidence of mitochondrial dysfunction and oxidative stress precedes the deposition of characteristic amyloid beta-plaques during disease progression [7, 8]

Med Sci Res 1991;19:543-544

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Furthermore, a large number of genetic variants have been described in the UGT genes that are due to mutations in both the coding and regulatory regions of the genes[4853]

[DOI] [PubMed] [Google Scholar] 35.Copur S, Demiray A, Kanbay M

T1 has been used in Phase III clinical trials with more than 4,000 patients, dosed continuously for 6 to 12 months in chronic hepatitis B, hepatitis C, and immunodeficiency indications