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Answer and interpretation This is probably valproic acid induced hyperammonia encephalopathy (VHE) VHE has been reported secondary to acute VPA overdose and in chronic normal therapeutic dosing, presenting as an acute and profound decreased level of consciousness, with or without increased seizure activity this is usually in association with an increased serum ammonia level (Mock and Schwetschenau, 2008) Patients at higher risk for VHE include those with liver impairment, on multiple anticonvulsants, or have other causes of increased ammonia production

However, these needle-free alternatives each have limitations of their own

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This study therefore carries medical significance, as it may directly contribute to the development of therapeutic strategies for human obesity and metabolic disorders

249 In the brains of transgenic parkin-deficient mice, the inhibition of p53 caused by parkin deficiency increases XBP-1 expression, which in turn promotes DJ-1 expression by binding and transactivating the DJ-1 gene promoter

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