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[4] The primary scientist in the cited study reports that The major pathophysiological basis of CAD is atherosclerosis in association with varieties of immunometabolic disorders that can suppress oxytocin (OT) receptor (OTR) signaling in the cardiovascular system (CVS). Oxytocin exposure appears to overcome the drawback of reduced receptor density and helps maintain cardiac integrity

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Collectively, these data show that Rab27a regulates mVEGFR1 levels and suggest that it functions through modulation of receptor palmitoylation, likely via spatial regulation of DHHC3