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The condition arises from the toxic effects of excessive alcohol, whose metabolites disrupt cellular functions like protein homeostasis, lipid synthesis, redox equilibrium, culminating in hepatocyte death [186]

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It also regulates the activity of the respiratory complex, calcium influx, and intracellular secondary neurotransmitters [4], as well as mitochondrial permeability transition (mPT) and the opening of mitochondrial transition pores [1,2,3,9]

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Due to their small size and hydrophobic surface, GPX4s have the ability to react with complex lipids in membranes (19)