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studied the relationship between UA levels and the presence/progression of non-motor symptoms in de novo PD [92]

These actions contribute to maintaining nerve integrity, promoting axonal regrowth, and enhancing Schwann cell remyelination (Table 2)

In the kidney, we observed widespread disruption in energy metabolism and multiple molecules involved in glycolysis, gluconeogenesis, and the TCA cycle were depleted, indicating a more global disruption in energy metabolism than has been previously appreciated

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Oxidative stress, mitochondrial dysfunction, and genetic polymorphisms of the GCLC gene may contribute to this vulnerability

doi: 10.2147/DDDT.S426870 75