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This phenomenon of copper imbalance is part of the AD pathology, referred to as the CuAD hypothesis, which suggests that both copper excess and deficiency lead to neuronal damage and functional impairment (88)

Lakhani, P., Bhanderi, D., & Modi, C

lncRNA sensing of a viral suppressor of RNAi activates non-canonical innate immune signaling in Drosophila

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(2010) utilized IGF-1 and its analogs to investigate the relative contributions of PI3K/Akt versus MAPK/ERK signaling in IGF-1R-mediated cellular responses, demonstrating context-dependent pathway utilization across different cell types