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Myalgic encephalomyelitis/chronic fatigue syndrome: a comprehensive review
6.2 Neural damage mechanisms The molecular disruptions outlined above initiate a series of cascading effects, leading to neural damage through oxidative stress, excitotoxicity, and demyelination

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Neurons pretreated with Dihexa demonstrate significantly enhanced survival when exposed to reactive oxygen species, excitotoxic glutamate concentrations, or metabolic impairment compared to untreated controls

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