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doi: 10.1073/pnas.1600299113
Design, characterization, and in vitro cellular inhibition and uptake of optimized genistein-loaded NLC for the prevention of posterior capsular opacification using response surface methodology
2004), albeit at higher concentrations than primary cells (Raza and John 2015), they do so via caspase-mediated apoptosis, not necrosis (Jaeschke et al
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Diabetologia 65 , 21572171 (2022)
A lack of selenium also worsens T-2 toxin-induced damage and reduces preventive autophagy
