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5-amino-1MQ reverses this process, restoring cellular capacity for fatty acid oxidation while simultaneously reducing the availability of methylation resources needed for lipogenic gene expression

Together with the kinetic model and its full numerics of the expression derived for the doseresponse relation, an approximated form of the expression clarifies the role played by the individual biochemical processes and allows us to identify four distinct regimes for the downregulation that emerge from the balance between phosphorylation, dephosphorylation, and the cellular level of -arrestin.

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