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Endothelial dysfunction may be the key mechanism in the pathogenesis of MetS-associated ED, and focusing on vascular endothelial function in the corpus cavernosum (CC) of MetS-associated ED holds significant promise for the discovery of drug targets and addressing the therapeutic challenges of organic ED

rosea (5.1) and placebo (4.6)

Borcherding N, Brestoff JR (2023) The power and potential of mitochondria transfer

The secretion ability of heme can be improved by increasing the expression of heme exporter

An individual patient may not precisely follow these stages

GSTOs have a cysteine residue at the catalytic site in place of the characteristic tyrosine or serine