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Subsequent work undertaken by the same authors suggested that impaired glycolytic formation of pyruvate may lead to the reduced provision of oxidised substrate for the tricarboxylic acid (TCA) cycle [109]
& Ghadhban, R
doi: 10.1210/endo-103-1-281
On the one hand, a growing body of evidence points to autophagy deficiency in ASD [242, 244,245,246]
Similar results showing the link between mitochondrial and synaptic dysfunctions affecting synaptic transmission in animal models with 22q11.2 microdeletion were also obtained by other researchers [360, 361]
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