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Hypercapnic acidosis attenuates endotoxin-induced acute lung injury
The underlying mechanism is believed to be associated with estrogens influence on critical brainstem regions integral to neurocardiovascular regulation (54)
Within dopaminergic neurons of the SNpc, these pathways promote oxidative stress, impair mitochondrial dysfunction, and promote apoptotic signaling [13]
Winstein CJ, Stein J, Arena R, Bates B, Cherney LR, Cramer SC, et al
Neural Regeneration Research, 17(3), 482487.
L., & Taylor, S
