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-syn promotes lipid peroxidation and disrupts neuronal iron balance, while iron accumulation accelerates -syn fibrillation and oxidative stress

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Human clinical data is limited, and claims that outpace that evidence should be viewed skeptically

i.p) doses of THC resulted in improved cognitive function in old mice (24 and 18-month-old, respectively), whereas the same dose induced cognitive impairments in adult mice (2-months-old

The soluble protofibrils formed from -synuclein exert their cytotoxicity through disruption of mitochondrial function, promotion of autophagy, lysosomal impairment and loss in regulation of calcium homeostasis (Van Laar et al., 2020), the two primary reasons accounting for loss in cellular homeostasis

IFN--deficient mice have lower gene expressions of molecules including ICAM-1, VCAM-1, IL-1, IL-1, IL-6, TNF-, MCP-1 and iNOS after APAP administration compared with WT mice, and reduce disease susceptibility to APAP [111]