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Statin adverse effects in females is prevented by co-therapy with a source of -3 fatty acids Our data raised the possibility that preventing a reduction in DHA levels in female mice treated with statin might diminish adverse effects
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Clin Exp Immunol (2010) 162:1223
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We further demonstrate that activation of both mTOR-STAT3 and ERK-CREB pathways, induced by elevated circulation amino acids, is responsible for upregulation of Vgf and Gcg expression following glucagon receptor blockade
Nutricion Hospitalaria
