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This suggests a preferential targeting of fat metabolism rather than general catabolism

[3] The speed of response depends on multiple factors including the severity and duration of deficiency, the presence of neurological complications, and individual metabolic differences

Second, it conserves methyl groups by reducing the consumption of SAM, which has implications for DNA and histone methylation, key mechanisms in gene expression regulation

In mitochondria [edit] The metabolism of propionyl-CoA occurs in the mitochondria and requires Vitamin B 12 (as adenosylcobalamin) to make succinyl-CoA

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Women report similar results timelines and side effect profiles as men