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Increased flux of key cellular energy regulators, including NAD + and SAM, may potentially define the therapeutic mechanism-of-action of NNMT inhibitors

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They interact with molecular oxygen, generate free radicals to damage adjacent keratinocytes, and stimulate the production of inflammatory mediators in keratinocytes ( 3941 )

5-Amino-1MQ blocks that drain at the source, so more nicotinamide stays available to regenerate NAD+