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Early literature surrounding the stable gastric pentadecapeptide focused heavily on cytoprotection, vascular recruitment, intestinal integrity, inflammatory injury environments, and disruptions involving the brain-gut axis

The mechanisms are not competing, they are complementary in theory

Research suggests that individuals with autism often have lower glutathione levels, which may reduce the body's ability to detoxify heavy metals and manage oxidative stress[10]

To bridge the gap between local damage to the stomach mucosa and the general toxicity seen in other organs caused by alcohol and NSAIDs, further research is needed

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