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Description
In summary, the available evidence states a model in which PM 2.5 exposure is not a universal asthma risk factor but rather a conditionally harmful exposure whose effects are increased by specific genetic backgrounds, mostly involving detoxification capacity ( GSTP1, GSTM1 ) and the 17q1221 susceptibility locus
Bruggraber, A
A prospective cohort study
Norenberg MD
His time in the military instilled in him a deep sense of discipline and commitment that would guide him throughout his life
7b, c), consistent with the observations in our cell culture studies
