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Haematologica 84, 10501051

Pro-inflammatory cytokines drive the expression of these catabolic factors in chondrocytes through the activation of transcription factors such as HIF-2 32 and NF-B 79

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Researchers reported that ferroptosis inhibitors (Ferr-1 and DFO), an apoptosis inhibitor (Z-VAD-fmk), necrotizing apoptosis inhibitors (Nec-1 and Nec-2) and an autophagy inhibitor (CQ) were all unable to attenuate cell death induced by high SLC7A11expression after glucose starvation