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Future studies should integrate single-cell omics and organoid models to decode patient-specific mitochondrial dysfunction subtypes, enabling precise design of personalized therapies to bridge the gap from bench to bedside

Mitochondrial dysfunction and oxidative stress in aging and cancer

L22 Intranasal application of NPY and NPY1336 ameliorate disease pathology in R6/2 mouse model of huntingtons disease

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Code availability Computational code reacquired to reproduce the figures of the paper has been deposited in the Zenodo database under the accession number ( References Rice, G

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