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Description
Proximity-dependent proteomics of the Chlamydia trachomatis inclusion membrane reveals functional interactions with endoplasmic reticulum exit sites

These manifestations stem from mitochondrial dysfunction, often caused by mutations in genes related to mitochondrial function, leading to impaired energy production and widespread neurological and metabolic abnormalities

Am J Clin Nutr 1987;46:830-4

10.1080/10408398.2021.1924612 159 Sharifi-RadJ.RodriguesC

Peter de Keizer and his groundbreaking findings

Oxidative stress markers and mitochondrial antioxidant enzyme expression are increased in aggressive Hodgkin lymphomas[J]
