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The treated mice resisted diet-induced obesity, showing lower fat accumulation and better glucose control, which implies MOTS-c can counteract the negative effects of a high-fat diet on weight gain and insulin action

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BPC-157 complements these mechanisms through its activation of VEGFR2 and eNOS pathways that promote angiogenesis and improve endothelial function, with the peptide's nitric oxide generation supporting vasodilation and reducing thrombotic risk while its growth factor sensitization enhances vascular responses to ischemic conditions

Examines NNMT regulation in hepatic tissue and its relationship to fasting states and lipid metabolism, providing mechanistic context for NNMT inhibitor research