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Mitochondrial electron transport chain complex dysfunction in the colonic mucosa in ulcerative colitis

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doi: 10.15252/embr.202051981 91 YaliLJiaQBingZPengHXinchongSQiaoHet al

HP and IPF patient material was exclusively derived from lung explant material, representing end-stage disease

Crucially, this pronounced reduction in the soluble protein pool provides a plausible context for the unexpected decline in antioxidant enzyme activities observed in our study

When efferocytosis is impaired, and adipocyte apoptosis increases in morbid obesity, metabolic syndrome, and T2DM, apoptotic bodies undergo secondary necrosis, releasing DAMPs that amplify local inflammation and reinforce a self-sustaining loop between adipose remodeling, immune activation, and insulin resistance [967,968,969,970]