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Mitochondrial outer membrane protein fundc2 promotes ferroptosis and contributes to doxorubicin-induced cardiomyopathy

Hyperhomocysteinemia-Mediated Endoplasmic Reticulum Stress in Skeletal Muscle Dysfunction via JNK/pro-inflammatory Pathway

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Anyhow, preventing iron accumulation in tissues and cells by mediating the key molecules of iron metabolism (transferrin receptor, ferroportin, hepcidin, etc.) may be a novel direction for the treatment of SLE

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In addition to the NF-B pathway, the mitogen-activated protein kinase (MAPK) signaling cascade is also an important regulator of inflammatory responses in CNS microglia