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99 A recent study evaluating neoplasia risk in patients with LS treated with ICI revealed persistent risk of subsequent primary tumors and lower neoantigen load in subsequent tumors

On the evidence question: The absence of Phase III human trial data for compounds like BPC-157 and TB-500 is a genuine clinical limitation, not a technicality

Defining the dominant ROS source(s) and downstream transcriptional factors in this setting will provide important directions for future work

doi: 10.1083/jcb.201604085 47 MeftahA.HasegawaH.KantrowitzJ

Interaction between glutathione and apoptosis A decrease in cellular GSH concentration has long been reported to be an early event in the apoptotic cascade induced by death receptor activation [5], mitochondrial apoptotic signaling [110], and oxidative stress [62,111]

Of late, multiple transcription factors such as TFAP2C, p53, AP-1, STAT, Nrf2, HIF-1, and C/EBP have been linked with the transcriptional regulation of seleno-GPxs [8, 21,22,23,24]