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Although NF-B is well known for promoting inflammation, it also modulates ferroptosis by suppressing GPX4 and regulating LCN2 expression

Since T ex int /CTL-like cells persist and dominate during the middle stages of chronic viral infection (e.g., days 1430 post infection), an overall increase in glycolytic metabolism is observed compared to acute infection, where respiring memory T cells are primarily present at these time points

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The immune system is also affected by glutathione, with Treg cells helping to prevent autoimmune diseases.[ref] One pathway of estrogen metabolism involves glutathione[[ref] Heres a specific detox example: Mercury is a toxic heavy metal that is eliminated from the body in a glutathione-dependent manner