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Mechanistically, HGF inhibits inflammatory M1-like macrophage production of pro-inflammatory cytokines and chemokines (such as IL-1, IL-6, TNF, CXCL1, CXCL10, and CCL2) by disrupting nuclear factor NF-B signaling (Giannopoulou et al., 2008) through the enhancement of the heme oxygenase-1 (HO-1) transcriptional pathway and the inactivation of the GSK-3 pathway (Kamimoto et al., 2009a,b

In organ transplant, an infection after a kidney transplant can result in eventual organ rejection by the recipient owing to increased cytokine production by innate immune cells

under ROS-related stimulation, the phosphorylation of RelA (p65) modulates NF-B activity, influencing the expression of downstream genes involved in inflammation, immunity, and cell growth

[DOI] [PubMed] [Google Scholar] Heo, H

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Here are the complete dosage charts for the three most common alternative volumes