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Neutral ceramidase regulates breast cancer progression by metabolic programming of TREM2-associated macrophages
Following the degradation of CCMH and DCCMH, their constituents CHCA, Met, and Cu + were gradually liberated into the cancer cells
If the genes governing this regeneration become less accessible due to age-related chromatin condensation, gut function suffers
C/EBP confers dependence to fatty acid anabolic pathways and vulnerability to lipid oxidative stressinduced ferroptosis in FLT3-mutant leukemia
B.NguyenA.KhooK
