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Liang Y, Tang X, Zhang X, Cao C, Yu M, Wan M

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Targeting ferroptosis for the treatment of cognitive impairment Given the core pathogenic role of ferroptosis in various cognitive disorders, targeting this pathway has emerged as a highly attractive and novel therapeutic strategy

Both induced and spontaneous models of RA have been shown to benefit from helminthic therapy through decreasing inflammasome activity at the site of disease and the production of Th1 cytokines such as TNF, while increasing IL-4 and IgG1 production (Table 1)

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