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Iron chelators represent an effective intervention for patients with excessive TfR1 expression, binding circulating and intracellular iron for excretion via urine or bile, thereby reducing ferroptosis (Rodrigues de Morais and Gambero, 2019)
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Supported by: US Department of the Army grant DAMD17-98-1-8621 and the Office of Research and Development, Medical Research Service, Department of Veterans Affairs (GWR), Hawaii Community Foundation Grant 20071373 (FPB), NIH RO1 NS40302 (MJB), NIH R24 DA027318 (MJB/FPB), NIH U01 AG019349 (LRW), and NIH G12 RR003061 which supports the JABSOM histology/imaging core facility
In copper-based catalytic systems, sustained GSH-driven copper redox cycling can generate hydroxyl radicals (OH), causing acute oxidative damage and inducing apoptosis in cancer cells 183-185
[DOI] [PubMed] [Google Scholar] 273.Cryan JF, Oriordan KJ, Cowan CSM, Sandhu KV, Bastiaanssen TFS, Boehme M, et al
C.DhingraN.KumarR.et al (2008)
