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Cost-efficiency varies significantly

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This review elucidates the molecular mechanisms of ferroptosis in OA and its critical role in disease progression, exploring the impacts of iron metabolism dysregulation, lipid peroxidation, and the collapse of antioxidant defense systems on cartilage degeneration, synovial inflammation, and subchondral bone remodeling

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The results indicated that pretreatment with 10 M ERK1/2 inhibitor obviously antagonized, while pretreatment with 10 M JNK inhibitor and 10 M p38 inhibitor had no effect on, BPC-157-induced proliferation, migration, and tube formation