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2010;12 Suppl 2:116125

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Selective permeabilization of the host cell membrane of Plasmodium falciparum -infected red blood cells with streptolysin O and equinatoxin II

Meanwhile, NO levels elevated in G2 in response to pathogenic sporozoite phases that penetrated and inflamed cecal cells (99)

Protein misfolding and failure of clearance systems, including the autophagy-lysosome and ubiquitin-proteasome pathways, further compromise the cellular environment and lead to toxic intracellular accumulations [31, 32]