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Are diagnostic criteria for general joint hypermobility and benign joint hypermobility syndrome based on reproducible and valid tests

Interestingly, treatment of age-related macular degeneration (AMD) with SHLP2 restored normal levels of OXPHOS complex protein subunits, increased the mitochondrial DNA copy number, attenuated amyloid beta-induced cellular and mitochondrial toxicity, inducesd anti-apoptotic effects, and prevented the loss of viable cells and mitochondria [29]
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AMPK, as the primary regulator of cellular energy balance, improves metabolic health by promoting fatty acid oxidation and reducing fatty acid synthesis ( 4.2 Metabolic reprogramming and ketone body production IF induces a metabolic reprogramming state in the body, significantly increasing the production of ketone bodies (such as -hydroxybutyrate)

J Toxicol Sci 43:89100
