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N-Acetyl Transferases (NAT) This class of enzymes is responsible for the transfer of an acetyl group to convert aromatic amines or hydrazines to aromatic amides and hydrazides, which is significant for those taking pharmaceuticals such as isoniazid, hydralazine, and sulphonamides [83]

This methylation inhibits transcription factor binding and converts chromatin into a more condensed, transcriptionally inactive state, leading to FMRP deficiency and the manifestation of FXS symptoms (216)

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Other withdrawal signs are not apparent, but this is due mainly to the high lipophilicity of 9 -THC which causes the drug to accumulate in fatty tissues, allowing for slow release and continuous low-level receptor activation

Importantly, the behavioural effects of AAS appear to be modulated by individual personality characteristics, suggesting that not all users exhibit uniform psychological responses (59)