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Inflammation & Immunological Research Investigated for its broad anti-inflammatory properties, including modulation of pro-inflammatory cytokines, reduction of C-reactive protein (CRP), and attenuation of systemic inflammatory markers in metabolic and non-metabolic disease models, suggesting potential research applications beyond classical endocrinological contexts
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Pre-existing mitochondrial dysfunction due to aging, diabetes, or ischemia creates a permissive environment for cuproptosis by depleting GSH and reducing SOD2 activity, thereby compromising the organelle's ability to chelate copper and neutralize mtROS [145, 146]
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Strategic combinations can produce results exceeding any single treatment
