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[24] This complex is too large to cross the capillary endothelium freely, so it functions as a slowly releasing circulating reservoir: it prolongs the systemic presence of IGF-1 while simultaneously keeping the bulk of it biologically restrained until it is handed off to tissues

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Its bacteriostatic properties help mitigate microbial interference that could otherwise affect the validity and reproducibility of research outcomes

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Nephrotoxicity of S-(2-chloroethyl)glutathione in the Fischer rat: evidence for -glutamyltranspeptidase-independent uptake by the kidney