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To confirm that GCL activity wholly accounted for ROS down-modulation, we expressed (Fig

Wu and Meininger, 1993) (Figure 7)

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Often referred to as the master antioxidant, glutathione plays a pivotal role in many bodily functions, especially detoxification and cellular protection

The N-methyl-d-aspartate (NMDA) receptor-mediated excitatory procedure, which provides the intracellular Ca2+ increase necessary for nNOS activation, is another mechanism by which UCB-induced neuronal damage occurs

The glutathione synthesis pathway is destroyed, and the oxidative damage in the cells increases, which leads to apoptosis and inhibits tumor growth