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Early work focused on GHK-Cu as a wound-healing and anti-inflammatory peptide

It is now clear that oxidative damage, including that associated with small A42 oligomers, by targeting many aspects of brain cell structure and function, is fundamental to the pathogenesis and progression of AD

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[25] In the treatment of paracetamol (acetaminophen) overdose, acetylcysteine acts to maintain or replenish depleted glutathione reserves in the liver and enhance non-toxic metabolism of acetaminophen