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In contrast, in human atherosclerosis, SMCs migration from the arterial media occurs before lipid accumulation and in fact SMC make up almost 50% of the foam cells in the plaque lesion [108]

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This could suggest a regulatory bottleneck at the level of Sphingosine conversion or degradation in these mutants

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doi: 10.3390/ijms22126262 100 KrauDFariOSibiliaM