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Treatment with TF appeared to restore aspects of iron homeostasis and reduce oxidative stress, mitochondrial damage, inflammation, complement activation, and ferroptosis in this model

Costiniuk CT, Brunet L, Rollet-Kurhajec KC, Cooper CL, Walmsley SL, Gill MJ, et al

doi: 10.1371/journal.pone.0220123 47 MitraM.AgarwalP.RoyS

In the absence of a demonstrated functional role for this site, it has been suggested that HbF phenotype is modified by cis -linked elements located elsewhere in the -globin cluster and in linkage disequilibrium with this SNP [28], [29]